N- terminal BET bromodomain inhibitors disrupt a BRD4-p65

terminal bet Discussion. This study describes a mechanism by which inhibition of a specific BET bromodomain reduces NOS2 transcription and β-cell dysfunction. We investigated the ability of BET bromodomain small molecule inhibitors to reduce the β-cell response to the proinflammatory cytokine interleukin 1 beta (IL-1

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